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    • Tenascin-C drives persistence of organ fibrosis.
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Tenascin-C drives persistence of organ fibrosis.

Tenascin-C drives persistence of organ fibrosis. Nat Commun. 2016 06 03; 7:11703.

View in: PubMed

subject areas
  • Adult
  • Aged
  • Animals
  • Case-Control Studies
  • Cell Differentiation
  • Cells, Cultured
  • Collagen
  • Disease Models, Animal
  • Female
  • Fibroblasts
  • Fibrosis
  • Gene Expression Regulation
  • Humans
  • Lung
  • Male
  • Mice
  • Middle Aged
  • Myofibroblasts
  • Scleroderma, Systemic
  • Signal Transduction
  • Skin
  • Tenascin
  • Toll-Like Receptor 4
  • Up-Regulation

authors with profiles
  • Carol Feghali-Bostwick