Cyclic AMP Response Element-Binding Protein
"Cyclic AMP Response Element-Binding Protein" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A protein that has been shown to function as a calcium-regulated transcription factor as well as a substrate for depolarization-activated CALCIUM-CALMODULIN-DEPENDENT PROTEIN KINASES. This protein functions to integrate both calcium and cAMP signals.
Descriptor ID |
D017362
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MeSH Number(s) |
D12.776.260.108.184 D12.776.930.127.184
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Concept/Terms |
Cyclic AMP Response Element-Binding Protein- Cyclic AMP Response Element-Binding Protein
- CREB Protein
- DNA-Binding Protein, Cyclic AMP-Responsive
- DNA Binding Protein, Cyclic AMP Responsive
- cAMP Response Element-Binding Protein
- cAMP Response Element Binding Protein
- Cyclic AMP-Responsive DNA-Binding Protein
- Cyclic AMP Responsive DNA Binding Protein
|
Below are MeSH descriptors whose meaning is more general than "Cyclic AMP Response Element-Binding Protein".
Below are MeSH descriptors whose meaning is more specific than "Cyclic AMP Response Element-Binding Protein".
This graph shows the total number of publications written about "Cyclic AMP Response Element-Binding Protein" by people in this website by year, and whether "Cyclic AMP Response Element-Binding Protein" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1994 | 1 | 0 | 1 |
1995 | 1 | 0 | 1 |
1999 | 0 | 2 | 2 |
2000 | 1 | 0 | 1 |
2001 | 0 | 1 | 1 |
2002 | 1 | 0 | 1 |
2004 | 1 | 0 | 1 |
2005 | 2 | 0 | 2 |
2006 | 0 | 1 | 1 |
2007 | 3 | 2 | 5 |
2008 | 1 | 0 | 1 |
2009 | 1 | 1 | 2 |
2010 | 0 | 1 | 1 |
2011 | 2 | 0 | 2 |
2012 | 1 | 0 | 1 |
2013 | 0 | 1 | 1 |
2014 | 0 | 1 | 1 |
2016 | 1 | 0 | 1 |
2017 | 1 | 1 | 2 |
2018 | 0 | 1 | 1 |
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Below are the most recent publications written about "Cyclic AMP Response Element-Binding Protein" by people in Profiles.
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Biphasic effect of abstinence duration following cocaine self-administration on spine morphology and plasticity-related proteins in prelimbic cortical neurons projecting to the nucleus accumbens core. Brain Struct Funct. 2019 Mar; 224(2):741-758.
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A CREB3-regulated ER-Golgi trafficking signature promotes metastatic progression in breast cancer. Oncogene. 2018 03; 37(10):1308-1325.
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Cyclic nucleotide signaling changes associated with normal aging and age-related diseases of the brain. Cell Signal. 2018 Jan; 42:281-291.
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Distinct Roles of CREB Within the Ventral and Dorsal Hippocampus in Mediating Nicotine Withdrawal Phenotypes. Neuropsychopharmacology. 2017 Jul; 42(8):1599-1609.
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Short-term effects of an endotoxin on substantia nigra dopamine neurons. Brain Res. 2014 Apr 04; 1557:164-70.
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Short and long access to cocaine self-administration activates tyrosine phosphatase STEP and attenuates GluN expression but differentially regulates GluA expression in the prefrontal cortex. Psychopharmacology (Berl). 2013 Oct; 229(4):603-13.
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Endoplasmic reticulum-tethered transcription factor cAMP responsive element-binding protein, hepatocyte specific, regulates hepatic lipogenesis, fatty acid oxidation, and lipolysis upon metabolic stress in mice. Hepatology. 2012 Apr; 55(4):1070-82.
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The transcription factor cyclic AMP-responsive element-binding protein H regulates triglyceride metabolism. Nat Med. 2011 Jun 12; 17(7):812-5.
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A silent synapse-based mechanism for cocaine-induced locomotor sensitization. J Neurosci. 2011 Jun 01; 31(22):8163-74.
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Stimulated neuronal expression of brain-derived neurotrophic factor by Neurotropin. Mol Cell Neurosci. 2010 Nov; 45(3):226-33.