"Repressor Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proteins which maintain the transcriptional quiescence of specific GENES or OPERONS. Classical repressor proteins are DNA-binding proteins that are normally bound to the OPERATOR REGION of an operon, or the ENHANCER SEQUENCES of a gene until a signal occurs that causes their release.
| Descriptor ID |
D012097
|
| MeSH Number(s) |
D12.776.260.703 D12.776.930.780
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Repressor Proteins".
Below are MeSH descriptors whose meaning is more specific than "Repressor Proteins".
This graph shows the total number of publications written about "Repressor Proteins" by people in this website by year, and whether "Repressor Proteins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 1 | 0 | 1 |
| 1995 | 3 | 0 | 3 |
| 1996 | 6 | 0 | 6 |
| 1997 | 1 | 1 | 2 |
| 1998 | 6 | 1 | 7 |
| 1999 | 1 | 1 | 2 |
| 2000 | 5 | 2 | 7 |
| 2001 | 3 | 1 | 4 |
| 2002 | 3 | 0 | 3 |
| 2003 | 6 | 1 | 7 |
| 2004 | 3 | 3 | 6 |
| 2005 | 4 | 2 | 6 |
| 2006 | 1 | 1 | 2 |
| 2007 | 4 | 5 | 9 |
| 2008 | 4 | 2 | 6 |
| 2009 | 5 | 6 | 11 |
| 2010 | 3 | 2 | 5 |
| 2011 | 0 | 3 | 3 |
| 2012 | 2 | 2 | 4 |
| 2013 | 2 | 4 | 6 |
| 2014 | 3 | 6 | 9 |
| 2015 | 4 | 2 | 6 |
| 2016 | 3 | 2 | 5 |
| 2017 | 4 | 1 | 5 |
| 2018 | 3 | 2 | 5 |
| 2019 | 3 | 1 | 4 |
| 2020 | 2 | 1 | 3 |
| 2021 | 1 | 0 | 1 |
| 2022 | 0 | 2 | 2 |
| 2023 | 1 | 0 | 1 |
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Below are the most recent publications written about "Repressor Proteins" by people in Profiles.
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CITED2 is a conserved regulator of the uterine-placental interface. Proc Natl Acad Sci U S A. 2023 01 17; 120(3):e2213622120.
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Primary Spindle Cell Sarcoma of the Lung with MGA::NUTM1 Fusion: An Extremely Rare Case of a Potentially Emerging Entity and Review of the Literature. Int J Surg Pathol. 2022 Dec; 30(8):931-938.
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Expansion of Candidate HPV-Specific T Cells in the Tumor Microenvironment during Chemoradiotherapy Is Prognostic in HPV16+ Cancers. Cancer Immunol Res. 2022 02; 10(2):259-271.
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Tribbles Homolog 3 Mediates the Development and Progression of Diabetic Retinopathy. Diabetes. 2021 08; 70(8):1738-1753.
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HPV-inactive cell populations arise from HPV16-transformed human keratinocytes after p53 knockout. Virology. 2021 02; 554:9-16.
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Intrinsically Disordered Regions of the DNA-Binding Domain of Human FoxP1 Facilitate Domain Swapping. J Mol Biol. 2020 09 04; 432(19):5411-5429.
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Genomic influences on self-reported childhood maltreatment. Transl Psychiatry. 2020 01 27; 10(1):38.
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Targeting interferon signaling and CTLA-4 enhance the therapeutic efficacy of anti-PD-1 immunotherapy in preclinical model of HPV+ oral cancer. J Immunother Cancer. 2019 09 18; 7(1):252.
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PTEN Methylation by NSD2 Controls Cellular Sensitivity to DNA Damage. Cancer Discov. 2019 09; 9(9):1306-1323.
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Two Distinct E2F Transcriptional Modules Drive Cell Cycles and Differentiation. Cell Rep. 2019 06 18; 27(12):3547-3560.e5.